منابع مشابه
Update on Plant Cell Cycle Control Switching the Cell Cycle. Kip-Related Proteins in Plant Cell Cycle Control
During the development of multicellular organisms, many different cell types are created. These display characteristic cell cycle programs that can radically change during the organism’s lifetime (Jakoby and Schnittger, 2004; Fig. 1). Usually, in younger and less differentiated tissues, a proliferative cell cycle mode occurs. In this mitotic cell cycle program, DNA replication (also known as th...
متن کاملCell cycle control of BRCA2.
Identifying the conditions and kinetics of the induction of BRCA2 gene expression may implicate roles for the function of the tumor suppressor gene. In this study, expression of BRCA2 mRNA is shown to be regulated by the cell cycle and associated with proliferation in normal and tumor-derived breast epithelial cells. Cells arrested in G(0) or early G1 contained low levels of BRCA2 mRNA. After r...
متن کاملCell Cycle: Principles of Control
tion at the end of each case. For students desiring a more in-depth review, the analysis section of each case provides a great and more detailed overview. An extra index organized by disorder name in the back also allows the reader to focus on particular aspects of internal medicine. Details such as this make it obvious that the authors have tried to cater to the varying needs of their readers,...
متن کاملAdenovirus and cell cycle control.
Adenovirus infection of quiescent cells induces transition from G0 or G1 into the S phase of the cell cycle and allows cellular proliferation. This is beneficial for the virus since cells in S phase provide optimal conditions for viral replication. Adenovirus E1A, E1B and E4 gene products contribute to cell cycle deregulation. E1A proteins inactivate the pRb checkpoint, allowing the E2F transcr...
متن کاملذخیره در منابع من
با ذخیره ی این منبع در منابع من، دسترسی به آن را برای استفاده های بعدی آسان تر کنید
ژورنال
عنوان ژورنال: Current Opinion in Microbiology
سال: 2016
ISSN: 1369-5274
DOI: 10.1016/j.mib.2016.01.010